Bacterial Endotoxins: Basic Science to Anti-Sepsis Stratetgies

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This book brings together the most recent basic and clinical research on the molecular physiology of endotoxin pathology. Avenues for the development of new treatments, especially immunotherapies, are presented throughout.
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Overview

This book brings together the most recent basic and clinical research on the molecular physiology of endotoxin pathology. Avenues for the development of new treatments, especially immunotherapies, are presented throughout.
Read More Show Less

Product Details

Table of Contents

Contributors
Preface
Acknowledgments
The Receptor(s) for Endotoxin on Mammalian Cells 3
A Case for an Endotoxic Conformation 17
LPS Binding Protein and CD14 in the LPS-Dependent Activation of Cells
The Bactericidal/Permeability-Increasing Protein of Neutrophils Is a Potent Antibacterial and Anti-Endotoxin Agent In Vitro and In Vivo
Septin, an Activity in Plasma That Enables CD14-Dependent Recognition of LPS 53
Interaction Between CD14, Lipopolysaccharide-Binding Protein and Bactericidal/Permeability-Increasing Protein During Lipopolysaccharide-Induced Cell Activation 59
Biological Activity of Lipid A Partial Structures
Anti-Lipopolysaccharide Core Antibodies 85
Lipopolysaccharide-Antagonizing Effects of Diphosphoryl Lipid A From Rhodobacter sphaeroides (Rs-DPLA) 95
Prophylactic Use of Monophosphoryl Lipid A in Patients at Risk for Sepsis 107
Rabbit CAP18-Derived Peptides Inhibit Gram-Negative and Gram-Positive Bacteria 125
Phosphatidyl-Inositol-Linked CD14 Is Involved in Priming of Human Neutrophils by Lipopolysaccharide (LPS), but Not in the Inactivation of LPS 137
Endotoxin-Binding Synthetic Peptides With Endotoxin-Neutralizing, Antibacterial and Anticoagulant Activities 147
Hemoglobin: A Newly Recognized Binding Protein for Bacterial Endotoxins (LPS) 161
Studies on the Inflammatory-Coagulant Axis in the Baboon Response to E. coli: Regulatory Roles of Proteins C, S, C4bBP, and of Inhibitors of Tissue Factor 175
The Contact System and Sepsis 195
Inhibition of Extrinsic Coagulation Activation in Endotoxemia: Therapeutic Implications 215
Experimental and Clinical Evidence of Leukocyte Activation in Trauma and Sepsis 221
Endotoxin-Induced Coagulation Activation and Protein C Reduction in Meningococcal Septic Shock 247
Efficacy of a Recombinant Amino Terminal Fragment of Bactericidal/Permeability-Increasing Protein in Rodents Challenged With LPS or E. coli Bacteria 255
Pulmonary Injury Following Sepsis 265
Platelet-Activating Factor Antagonists as Therapeutic Strategy in Sepsis 277
Cytokine-Neutralizing Strategies in Experimental Sepsis 295
LPS, recIL1, and Smooth Muscle Cell-IL1 Activate Vascular Cells by Specific Mechanisms 309
Pentoxifylline Modulates Cytokine Responses in a Sepsis Model 323
Initial Studies on the Administration of C1-Esterase Inhibitor to Patients With Septic Shock or With a Vascular Leak Syndrome Induced by Interleukin-2 Therapy 335
Gene Therapy for Inflammatory Diseases 361
Regulation of the TNF[alpha] Gene 367
Endotoxin and the Release of Tumor Necrosis Factor Receptors 383
Participation of IFN-[gamma] in the Pathogenesis of LPS Lethality 399
Sepsis Criteria, and Selection of Patients for Immunotherapy 407
The Protective Role of Interleukin-10 in Endotoxin Shock 417
The Role of Tumor Necrosis Factor in Systemic Inflammatory Responses in Primate Endotoxemia 425
Cost-Effectiveness of HA-1A Treatment for Patients With Sepsis 435
Summary 445
Index 451
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