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"This work provides the most current and compelling information about nonverbal learning disabilities (NLD). Dr. Rourke has succeeded in bringing together international authorities to explicate issues relevant to the etiology, diagnosis, manifestation, developmental course, and treatment of the NLD syndrome across a number of pediatric neurological diseases and disorders. The comprehensive discussions of the white matter model are state-of-the-art and provide the first definitive classification of the cognitive, linguistic, perceptual, motor, and sensory system sequelae of the NLD syndrome. This book is a major contribution to the literature and should be required reading for researchers and clinicians." --G. Reid Lyon, Ph.D., National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland
"A unique volume presenting up-to-date reviews of a variety of genetic and acquired neurodevelopmental syndromes. Each chapter is organized around the central theme of Rourke's current model of nonverbal learning disabilities. Ranging from rare developmental to more common childhood disorders, anatomical, developmental, neuropsychological and neurological are presented. Each disorder is then addressed as to its relevance to the Rourke model. This volume is an excellent resource for readers interested in a developmental approach to understanding the behavioral manifestations of the nonverbal learning disabilities." --Eileen B. Fennell, Ph.D., Professor of Clinical and Health Psychology and Neurology, University of Florida.
"Byron Rourke has been a pioneer in developing the concept of a syndrome of nonverbal disabilities and foremost in study of its manifestations. His ideas on how to integrate findings on brain abnormalities and on associated cognitive and behavioral malfunction are constructively provocative and controversial. This edited volume takes on the ambitious task of bringing together such apparently diverse disorders such as Asperger syndrome, Williams syndrome, hydrocephalus, fetal alcohol syndrome and traumatic brain injury within a model of which brain matter dysfunction, with the manifestations modified by amount of tissue damage and developmental timing. Not everyone will agree with the propositions put forward but all readers will find this impressively scholarly, but readable, review of the evidence and conceptual inferences usefully thought-provoking." --Michael Rutter, MD, FRS, Medical Research Council Child Psychiatry Unit, London