Algodystrophy: Diagnosis and Therapy of a Frequent Disease of the Locomotor Apparatus

Algodystrophy: Diagnosis and Therapy of a Frequent Disease of the Locomotor Apparatus

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Overview

Algodystrophy: Diagnosis and Therapy of a Frequent Disease of the Locomotor Apparatus by P. Doury, Y. Dirheimer, S. Pattin

It is with great pleasure and ,much interest that I accepted to write the foreword to this book by Paul Doury, Yves Dirheimer, and Serge Pattin on the subject of "algodystrophy." First, because I know the extent of their personal experience, from which they have selected the best for this book. Second, because it seemed to me that their detailed analysis of the numerous works on the subject, works which have been published all over the world and which provide diverse physiopathologic interpretations, would provide a comprehensive study meeting a real need. Algodystrophy, to adopt the term used by the authors, merits rheumatolog­ ists' careful attention. It is indeed a frequent condition and, as is now well known, occurs in the most varied etiologic circumstances; it is not solely posttraumatic, a notion on which diagnosis had long been based. This variable etiology suggests the complexity of algodystrophy's pathogenic mechanism.

Product Details

ISBN-13: 9783642679919
Publisher: Springer Berlin Heidelberg
Publication date: 11/19/2011
Edition description: Softcover reprint of the original 1st ed. 1981
Pages: 168
Product dimensions: 8.27(w) x 11.02(h) x 0.02(d)

Table of Contents

1 Historical Review.- 2 Why Study this Condition?.- 1 Symptomatology.- 1 General Data in Algodystrophy.- A. Clinical Syndrome.- B. Biochemical Syndrome.- C. Radiological Syndrome.- D. Scintigraphic Syndrome.- E. Clinical Course.- 2 Clinical Description.- 3 Topographic Forms.- A. Algodystrophy of the Upper Limb.- I. Onset.- II. First Stage.- 1. Involvement of Hand and Wrist.- 2. Involvement of the Shoulder.- III. Second Stage.- 1. Hand and Wrist.- 2. Shoulder.- 3. Elbow.- IV. Evolution.- V. Topographic Forms.- 1. Isolated Involvement of the Hand.- 2. Isolated Involvement of the Shoulder.- 3. Associated Involvement of the Elbow.- 4. Associated Involvement of the Sternoclavicular Articulation.- 5. Algodystrophy Localized to Two or Three Fingers of One Hand.- 6. Bilateral and Extensive Forms.- B. Algodystrophy of the Lower Limbs.- I. Algodystrophy of the Foot.- II. Algodystrophy of the Knee.- 1. The Classical Form.- 2. Partial Algodystrophy.- 3. Highly Localized Algodystrophy.- III. Algodystrophy of the Hip.- 1. General Data.- 2. Clinical Signs.- 3. Clinical Course.- C. Algodystrophy of the Spine.- D. Other Localizations.- 2 Complementary Investigations.- 1 Biochemistry.- A. Nonspecific Indicators of Inflammation.- B. Calcium and Phosphorus Levels in Blood and Urine.- C. Calcitonin Test.- D. Hydroxyproline in the Urine.- E. Parathormone.- F. HL-A Antigen.- G. Synovial Fluid.- H. Muscular Enzymes.- Predisposition to Algodystrophy in Certain Preexisting Metabolic Disturbances.- 1. Diabetes Mellitus.- 2. Hyperlipidemia.- 2 Radiology.- A. Techniques.- I. Magnification Radiology.- II. Tomography.- III. Xeroradiography.- IV. Electron Radiography.- V. Logetronography.- B. Involvement of Soft Tissues.- C. Involvement of Bone.- I. Elementary Radiographic Signs.- 1. Osteopenia.- 2. Subchondral Lysis.- 3. Resorption of Cortical Bone.- 4. Juxta-articular Erosions.- II. Association of Primary Lesions Producing Different Types of Image.- III. Evolution Features.- 1. Early Demineralization.- 2. Evaluation of the Radiologic Intensity of Algodystrophy.- 3. Classification into Stages.- 4. Forms Without Bone Demineralization.- D. Topographic Forms.- I. Partial Algodystrophy.- II. Highly Localized or Parcellar Algodystrophy.- III. Algodystrophy of the Upper Limb.- IV. Algodystrophy of the Foot.- V. Algodystrophy of the Knee.- VI. Algodystrophy of the Hip.- VII. Algodystrophy of the Spine.- E. Articular Involvement.- F. Arthrography...- G. Arteriography.- H. Pertrochanteral and Intra-femoral-head Phlebography.- 3 Scintigraphy.- A. Bone Scanning.- I. Technique.- II. Results.- 1. Increased Uptake.- 2. Iso and Hypofixation.- 3. Usefulness of Bone Scanning for Diagnosis of Algodystrophy.- 4. Limits of Bone Scanning.- 5. Significance of Scintigraphic Data.- B. Isotopic Exploration of the Vascular Sector.- C. Isotopic Exploration of the Interstitial Area.- D. Joint Scanning.- 4 Densitometry.- 5 Thermography.- 6 Vascular Investigations.- A. Oscillometry.- B. Capillaroscopy.- C. Doppler Effect.- 7 Electromyography.- 8 Pathology.- A. Synovia.- B. Cartilage.- C. Bone.- I. Macroscopy.- II. Microscopy.- III. Electron Microscopy.- IV. Microradiography.- V. Static and Dynamic Histomorphometry.- VI. Attempt at a Histodynamic Interpretation of the Algodystrophic Mechanism.- D. Periarticular Tissues.- I. Capsule.- II. Aponeuroses, Bursae and Tendons.- III. Muscles.- IV. Cutaneous Tissues.- V. Vessels.- VI. Nerves.- 3 Clinical Forms.- 1 Topographic Forms.- 2 Evolutive Forms.- A. Initial Plurifocal Forms.- I. Patent Forms.- 1. In the Upper Limb.- 2. In the Lower Limb.- 3. Simultaneous Involvement of the Four Limbs.- II. Latent Forms.- B. Secondarily Extensive Forms.- C. True Recurrent Forms.- D. Partial Forms.- I. Radial Forms.- II. Zonal Forms.- E. Par cellar Forms.- 3 Incomplete Forms.- A. Clinical.- B. Radiological.- C. Scintigraphic.- 4 Atypical Forms.- A. Hyperinflammatory Forms.- B. Pseudophlegmonous Forms.- C. Fibrotic Forms (Pseudo-Dupuytren).- D. Pseudosclerodermic Forms.- 4 Differential Diagnosis.- 1 Inflammatory Process.- A. Synovitis.- B. Phlebitis.- 2 Infection.- A. Osteitis.- B. Abscesses.- C. Septic Arthritis.- 3 Tumors.- A. Benign Tumors.- B. Malignant Tumors.- 4 Bone Necrosis.- 5 Arthrosis.- 6 Miscellaneous.- A. Fatigue Fractures.- B. Chondromatosis — Osteochondromatosis.- C. Dupuytren’s Contracture.- D. Dupuytren’s Disease of the Foot.- E. Scleroderma.- F. Osteolysis.- I. Idiopathic Osteolysis.- II. Idiopathic Acro-osteolysis.- III. Secondary Osteolysis.- G. Osteoporosis Following Immobilization.- H. Posttraumatic Paget’s Disease.- I. Sprains.- K. Somatic Conversion Syndromes: Hysteria.- 5 Positive Criteria.- 5 Positive Criteria.- Clinical Criteria.- Radiological Criteria.- Biochemical Criteria.- Scintigraphic Criteria.- Criteria Related to the Clinical Course and to the Treatment.- 6 Etiology.- 6 Etiology.- A. General Etiologic Circumstances.- I. Age.- II. Sex.- B. Favoring Circumstances.- I. Traumatism.- 1. The Severity of the Causal Traumatism.- 2. The Nature of the Traumatism.- 3. The Site of the Traumatism.- 4. Algodystrophy of the Limbs and Immobilization.- II. Nontraumatic Affections of the Locomotor Apparatus...- III. Diseases of the Nervous System.- 1. Involvement of the Peripheral Nerves.- 2. Diseases of the Central Nervous System.- IV. Cardiovascular Diseases.- V. Pleuropulmonary Diseases.- VI. Hyperthyroidism.- VII. Diabetes Mellitus.- VIII. Hyperlipidemia.- IX. Iatrogenic Algodystrophies.- 1. Phenobarbitals.- 2. Tuberculostats Ill.- 3. Pregnancy.- X. Other Etiologic Factors.- XI. Apparently Primary Algodystrophies.- XII. Etiologic Factors in Our 250 Cases of Algodystrophy.- 7 Physiopathology.- 7 Physiopathology.- 8 Treatment.- 1 Chemotherapy.- A. Analgesics.- B. Nonsteroid Anti-Inflammatory Drugs.- C. Systemic Corticosteroids.- D. Local Corticosteroids.- E. Therapy Aimed at Vasodilatation.- I. Systemic Treatment.- 1. Alpha-blockers.- 2. Ganglioplegics.- 3. Postganglionic Blockers.- II. Local Treatment.- F. Griseofulvin.- G. Beta-blockers.- H. Calcitonin.- 2 Physical Treatment.- A. Massage.- B. Hydrotherapy, Crenotherapy, Thalassotherapy.- C. Kinesitherapy and Rehabilitation.- D. Electrotherapy.- E. Roentgen therapy.- F. Acupuncture.- 3 Surgery.- 4 Management of Algodystrophies.- 5 Prevention.- 9 Clinical Course and Prognosis.- 1 Clinical Course.- A. Clinical Course of Untreated Algodystrophy.- B. Clinical Course of Treated Algodystrophy.- 2 Prognosis.- 10 Medicolegal Considerations.- 10 Medicolegal Considerations.- Causality.- Past Status.- Duration of Total Transient Disability.- Date of Consolidation.- Fixation of Permanent and Partial Disability.- The Pretium Doloris.- References.

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